Introduction
Spontaneous coronary artery dissection (SCAD) is a rare entity. It consists of the separation of the several layers of the coronary arteries, with or without an intimal flap1-3. In other words, at this time two physiopathological processes are identified: the first one is the rupture of the intimal and often the medial layers, as well as the engulfment of blood in that false lumen witch then compress the true lumen leading to a ischemia of the coronary artery's territory. The second one implies a probable rupture of the vasa vasorum, which leads to an intra-­‐‑mural hematoma, without rupture of the coronary artery's layers. In that situation there is no communication between the true and the false lumen1,2,4,5. Some studies include SCAD patients with underlying artheromtous disease, but commonly SCAD is considered as a synonym for a non-­‐‑ atherosclerotic coronary arterie's dissection1-4,6.